However, the GH/IGF-I axis in these disease states generally resets itself following the resolution of the inflammatory trigger (65)
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Cellular Signalling 13 , 221231, doi:10.1016/S0898-6568(01)00139-5 (2001)
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These results support the hypothesis that the T2-induced 15LO1 pathwayinduced alteration of intracellular redox balance enhances T2 inflammatory responses ex vivo
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